[1]  Taylor G I, Corlett R J, Caddy C M, et al. An anatomic review of the delay phenomenon: II. Clinical applications.[J]. Plastic Reconstr Surg, 1992, 89(3): 408-418.
[2]  张琰，毛军胜，王炳臣，等. 血管内皮生长因子基因治疗与皮瓣延迟术对大鼠腹壁轴型皮瓣成活的影响[J]. 中华整形外科杂志, 2008, 24(1):39-41.
[3]  李养群，周传德，杨明勇，等. 岛状颞浅血管颈部预制扩张皮瓣修复面部软组织缺损[J]. 中国修复重建外科杂志, 2005, 19(2): 130-132.
[4]  吴建明，林子豪，刘麒. 热应激预处理对皮瓣缺血再灌注损伤的影响及机制[J]. 中华整形外科杂志, 1999, 15(5): 351-353.
[5]  周达，李青峰. 预处理在预防皮瓣缺血性损伤中的研究进展[J]. 组织工程与重建外科杂志, 2011, 7(4): 238-240.
[6]  范启申，周祥吉，李庆喜，等. 吻合一组血管的联合皮瓣移植临床应用探讨[J]. 中华显微外科杂志, 2005, 28(3): 200-202.
[7]  刘丽，张建宁. 内皮祖细胞的生物学特征及其在疾病研究中的意义[J]. 中华医学杂志, 2007, 87(7): 501-504.
[8]  朱华锋，汪春兰，赵宇. VEGF和FGF-2在血管生成中的协同作用研究进展[J]. 中华整形外科杂志, 2006, 22(1): 72-75.
[9]  许逸飞. TLR2基因在新生血管形成中的作用[D]. 浙江大学, 2012.
[10] 杨志峰，杨清玲，陈昌杰. 趋化因子SDF-1与受体CXCR4的研究进展[J]. 分子诊断与治疗杂志, 2011, 3(1): 58-61.
[11] Barnucz E, Veres G, Heged?s P, et al. Prolyl-hydroxylase inhibition preserves endothelial cell function in a rat model of vascular ischemia reperfusion injury[J]. J Pharmacol Exp Ther, 2013, 345(1): 25-31.
[12] Döring Y, Pawig L, Weber C, et al. The CXCL12/CXCR4 chemokine ligand/receptor axis in cardiovascular disease[J]. Front Physiol, 2014, 5(5): 212.
[13] Domanska UM, Kruizinga RC, Nagengast WB, et al. A review on CXCR4/CXCL12 axis in oncology: no place to hide[J]. Eur J Cancer,2012,49(1): 219-230.
[14] 刘泽隆，范红霞，王淑芳. 对vasculogenesis和angiogenesis中文翻译的探讨[J]. 中国科技术语, 2007, 9(4): 53-54.
[15] Risau W, Lemmon V. Changes in the vascular extracellular matrix during embryonic vasculogenesis and angiogenesis[J]. Dev Biol, 1988, 125(2):441-450.
[16] Asahara T, Murohara T, Sullivan A, et al. Isolation of putative progenitor endothelial cells for angiogenesis[J]. Science, 1997, 275(5302): 964-967.
[17] Wang JF, Park IW, Groopman JE. Stromal cell-derived factor-1alpha stimulates tyrosine phosphorylation of multiple focal adhesion proteins and induces migration of hematopoietic progenitor cells: roles of phosphoinositide-3 kinase and protein kinase C[J]. Blood, 2000, 95(8): 2505-2513.
[18] Tysseling VM, Mithal D, Sahni V, et al. SDF1 in the dorsal corticospinal tract promotes CXCR4+ cell migration after spinal cord injury[J]. J Neuroinflammation, 2011, 8(3): 1-8.
[19] Schober A, Karshovska E, Zernecke A, et al. SDF-1alpha-mediated tissue repair by stem cells: a promising tool in cardiovascular medicine [J] ?  Trends in Cardiovasc Med, 2006,16(4): 103-108.
[20] Salcedo R, Wasserman K, Young HA, et al. Vascular endothelial growth factor and basic fibroblast growth factor induce expression of CXCR4 on human endothelial cells: in vivo neovascularization induced by stromal-derived factor-1α[J].  Am J Pathol, 1999, 154(4): 1125-1135.
[21] Takemoto Y, Li TS, Kubo M, et al. The mobilization and recruitment of C-Kit+ cells contribute to wound healing after surgery[J]. Plos One, 2012, 7(11): e48052.
[22] Paganessi LA, Walker AL, Tan LL, et al. Effective mobilization of hematopoietic progenitor cells in G-CSF mobilization defective CD26-/- mice through AMD3100-induced disruption of the CXCL12-CXCR4 axis [J]. Exp Hematol, 2011, 39(3): 384-390.
[23] Suzuki A, Osanai T, Tanaka M, et al. Coupling factor 6 attenuates CXCR4 expression through the HIF-1α and c-Src pathways and promotes endothelial apoptosis and inflammation[J]. Hypertens Res, 2014, 37(8):708-715.
[24] Tachibana K, Hirota S, Iizasa H, et al. The chemokine receptor CXCR4 is essential for vascularization of the gastrointestinal tract[J]. Nature,1998, 393(6685): 591-594.
[25] Sk A, Pm H. Delayed supramalleolar flap--an innovative technique for enhanced viability[J]. J Ayub Med Coll Abbottabad, 2005, 17(3): 76-79.
[26] Program NT. Final report on carcinogens background document for formaldehyde[J]. Rep Carcinog Backgr Doc, 2010, (10-5981): i-512.
[27] Gallagher KA, Liu ZJ, Xiao M, et al. Diabetic impairments in NO-mediated endothelial progenitor cell mobilization and homing are reversed by hyperoxia and SDF-1 alpha[J]. J Clin Invest, 2007, 117(5): 1249-1259.
[28] Liu X, Liang F, Yang J, et al. Effects of stromal cell derived factor-1 and CXCR4 on the promotion of neovascularization by hyperbaric oxygen treatment in skin flaps[J]. Mol Med Rep, 2013, 8(4): 1118-1124.
[29] Zgraggen S, Huggenberger R, Kerl K, et al. An important role of the SDF-1/CXCR4 axis in chronic skin inflammation[J]. Plos One, 2014, 9(4): e93665.