[1] Porter C, Tompkins R G, Finnerty C C, et al. The metabolic stress response to burn trauma: current understanding and therapies [J]. Lancet, 2016, 388(10052): 1417-1426. DOI: 10.1016/s0140-6736(16)31469-6.
[2] Stanojcic M, Abdullahi A, Rehou S, et al. Pathophysiological response to burn injury in adults [J]. Annals of surgery, 2018, 267(3): 576-584. DOI: 10.1097/sla.0000000000002097.
[3] Jeschke M G, Gauglitz G G, Kulp G A, et al. Long-term persistance of the pathophysiologic response to severe burn injury [J]. Plos one, 2011, 6(7): e21245. DOI: 10.1371/journal.pone.0021245.
[4] Schoettl T, Fischer I P, Ussar S. Heterogeneity of adipose tissue in development and metabolic function [J]. The Journal of experimental biology, 2018, 221(Pt Suppl 1): jeb162958. DOI: 10.1242/jeb.162958.
[5] Ikeda K, Maretich P, Kajimura S. The common and distinct features of brown and beige adipocytes [J]. Trends in endocrinology and metabolism, 2018, 29(3): 191-200. DOI: 10.1016/j.tem.2018.01.001.
[6] Biswas H M. Effects of α-(prazosin and yohimbine) and β-receptors activity on cAMP generation and UCP1 gene expression in brown adipocytes [J]. Journal of basic and clinical physiology and pharmacology, 2018, 29(5): 545-552. DOI: 10.1515/jbcpp-2017-0211.
[7] Wu J, Boström P, Sparks L M, et al. Beige adipocytes are a distinct type of thermogenic fat cell in mouse and human [J]. Cell, 2012, 150(2): 366-376. DOI: 10.1016/j.cell.2012.05.016.
[8] Knuth C M, Barayan D, Lee J H, et al. Subcutaneous white adipose tissue independently regulates burn-induced hypermetabolism via immune-adipose crosstalk [J]. Cell reports, 2024, 43(1): 113584. DOI: 10.1016/j.celrep.2023.113584.
[9] Yo K, Yu Y M, Zhao G, et al. Brown adipose tissue and its modulation by a mitochondria-targeted peptide in rat burn injury-induced hypermetabolism [J]. American journal of physiology Endocrinology and metabolism,2013, 304(4): E331-341. DOI: 10.1152/ajpendo. 00098. 2012.
[10]Patsouris D, Qi P, Abdullahi A, et al. Burn induces browning of the subcutaneous white adipose tissue in mice and humans [J]. Cell reports, 2015, 13(8): 1538-1544. DOI: 10.1016/j.celrep.2015.10.028.
[11]Abdullahi A, Chen P, Stanojcic M, et al. IL-6 Signal From the bone marrow is required for the browning of white adipose tissue post burn injury [J]. Shock,2017, 47(1): 33-39. DOI: 10.1097/shk.0000 000000000749.
[12]Saraf M K, Herndon D N, Porter C, et al. Morphological changes in subcutaneous white adipose tissue after severe burn injury [J]. Journal of burn care & research, 2016, 37(2): e96-103. DOI: 10.1097/bcr.0000000000000292.
[13]Kaur S, Auger C, Barayan D, et al. Adipose-specific ATGL ablation reduces burn injury-induced metabolic derangements in mice [J]. Clinical and translational medicine, 2021, 11(6): e417. DOI: 10.1002/ctm2.417.
[14]Barayan D, Vinaik R, Auger C, et al. Inhibition of lipolysis with acipimox attenuates postburn white adipose tissue browning and hepatic fat infiltration [J]. Shock, 2020, 53(2): 137-145. DOI: 10.1097/shk.0000000000001439.
[15]Raje V, Ahern K W, Martinez B A, et al. Adipocyte lipolysis drives acute stress-induced insulin resistance [J]. Scientific reports, 2020, 10(1): 18166. DOI: 10.1038/s41598-020-75321-0.
[16]Leitner B P, Huang S, Brychta R J, et al. Mapping of human brown adipose tissue in lean and obese young men [J]. Proceedings of the National Academy of Sciences of the United States of America, 2017, 114(32): 8649-8654. DOI: 10.1073/pnas.1705287114.
[17]Petruzzelli M, Schweiger M, Schreiber R, et al. A switch from white to brown fat increases energy expenditure in cancer-associated cachexia [J]. Cell metabolism, 2014, 20(3): 433-447. DOI: 10.1016/j.cmet.2014.06.011.
[18]Abdullahi A, Auger C, Stanojcic M, et al. Alternatively activated macrophages drive browning of white adipose tissue in burns [J]. Annals of surgery, 2019, 269(3): 554-563. DOI: 10.1097/sla.0000000000002465.
[19]Fischer K, Ruiz H H, Jhun K, et al. Alternatively activated macrophages do not synthesize catecholamines or contribute to adipose tissue adaptive thermogenesis [J]. Nature medicine, 2017, 23(5): 623-630. DOI: 10.1038/nm.4316.
[20]Qiu Y, Nguyen K D, Odegaard J I, et al. Eosinophils and type 2 cytokine signaling in macrophages orchestrate development of functional beige fat [J]. Cell, 2014, 157(6): 1292-1308. DOI: 10.1016/j.cell.2014.03.066.
[21]Vargovic P, Manz G, Kvetnansky R. Continuous cold exposure induces an anti-inflammatory response in mesenteric adipose tissue associated with catecholamine production and thermogenin expression in rats [J]. Endocrine regulations, 2016, 50(3): 137-144. DOI: 10.1515/enr-2016-0015.
[22]Knuth C M, Ricciuti Z, Barayan D, et al. Single-nuclei RNA profiling reveals disruption of adipokine and inflammatory signaling in adipose tissue of burn patients [J]. Annals of surgery, 2023, 278(6): e1267-e1276. DOI: 10.1097/sla.0000000000005880.
[23]Zhuang S, Chai J, Liu L, et al. Effect of celecoxib in treatment of burn-induced hypermetabolism [J]. Bioscience reports, 2020, 40(4): BSR20191607. DOI: 10.1042/bsr20191607.
[24]Vinaik R, Syanojcic M, Jeschke M G. NLRP3 inflammasome modulates post-burn lipolysis and hepatic fat infiltration via fatty acid synthase [J]. Scientific reports, 2018, 8(1): 15197. DOI: 10.1038/s41598-018-33486-9.
[25]Vinaik R, Barayan D, Abdullahi A, et al. NLRP3 inflammasome mediates white adipose tissue browning after burn [J]. American journal of physiology Endocrinology and metabolism, 2019, 317(5): e751-e759. DOI: 10.1152/ajpendo.00180.2019.
[26]Barayan D, Abdullahi A, Vinaik R, et al. Interleukin-6 blockade, a potential adjunct therapy for post-burn hypermetabolism [J]. FASEB journal, 2021, 35(5): e21596. DOI: 10.1096/fj.202100388R.
[27]Abdullahi A, Samadi O, Auger C, et al. Browning of white adipose tissue after a burn injury promotes hepatic steatosis and dysfunction [J]. Cell death & disease, 2019, 10(12): 870. DOI: 10.1038/s41419-019-2103-2.
[28]Zhang M, Yang P, Yu T, et al. Lytic cocktail: an effective method to alleviate severe burn induced hyper-metabolism through regulating white adipose tissue browning [J]. Heliyon, 2022, 8(3): e09128. DOI: 10.1016/j.heliyon.2022.e09128.
[29]Herndon D N, Rodriguez N A, Diaz E C, et al. Long-term propranolol use in severely burned pediatric patients: a randomized controlled study [J]. Annals of surgery,2012, 256(3): 402-411. DOI: 10.1097/SLA. 0b013e318265427e.
[30]Auger C, Knuth C M, Abdullahi A, et al. Metformin prevents the pathological browning of subcutaneous white adipose tissue [J]. Molecular metabolism,2019, 29: 12-23. DOI: 10.1016/j.molmet.2019. 08.011.
|
京ICP备08002354号-3
